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- A left anterior oblique view of the right coronary system, demonstrating a complete occlusion of the mid right coronary artery with thrombus.
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Moreover, a element of endothelial dysfunction has been proposed, which may contribute to irregular vessel reactivity. However, testosterone in physiologic doses may even be beneficial in patients with angina , have greater ischemic thresholds and improved high quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood circulate in males with established coronary artery illness . Therefore, an interaction of endogenous elements of the patient’s threat profile as well as the testosterone dosing could also be problems with whether opposed events happen.
Erythrocytosis has been reported as a potential impact of testosterone, which could be useful in patients with anemia. The best danger of erythrocytosis happens with injectable forms of testosterone in contrast with transdermal preparations, particularly in supraphysiologic doses. An increase of up to 44% of the baseline hematocrit has been reported .
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Rhoden EL, Morgentaler A. Risks of testosterone-replacement remedy and proposals for monitoring. Testosterone replacement in hypogonadal men with angina improves ischaemic threshold and high quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone improve human platelet thromboxane A2 receptor density and aggregation responses.
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We report a case of an acute ST-segment elevation myocardial infarction in a patient with conventional cardiac danger factors using supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this patient additionally had polycythemia, doubtless secondary to high-dose testosterone. The patient underwent successful percutaneous intervention of the best coronary artery. Physicians and sufferers should pay consideration to the clinical consequences of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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